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Regulating intra-cellular communication via prostaglandins has demonstrated superior diurnal IOP therapy for all other glaucoma prescription medications. Coleus forskohlii also regulates intra-cellular communication via c-AMP which mediates most of this effects of prostaglandin PGE 2 (see chart).
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Large diurnal fluctuations in IOP in day or higher consecutive days, such as those related to food sensitivities and allergies, are connected with an increased risk of for glaucoma progression above more traditional risk factors such as age, race and doing.
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PGF 2 derivatives are accustomed to medically lower IOP by affecting the FP receptor. These include latanoprost (Xalatan), travoprost (Travatan), bimatoprost (Lumigan) and unoprostone isopropyl (Rescula). Forskolin (Colforsin) works more than a complementary IOP-lowering but anti-inflammatory PGE 2 pathway from the c-AMP-mediated EP 2, EP 3 and EP 4 receptors.
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